LDL cholesterol, toxins and cardiovascular diseases – a reassessment

LDL cholesterol has been called "bad cholesterol" for decades. Textbooks consider it the primary cause of atherosclerosis and heart attacks. But this simple explanation doesn't stand up to critical scrutiny.

New findings show that LDL is not the cause of cardiovascular disease. Rather, it performs important protective and transport functions—and appears more as an epiphenomenon in the disease process: a side effect that points to deeper causes.

LDL as a binding carrier for toxins

LDL is a lipoprotein particle that transports cholesterol and fats in the blood. Its lipid-rich structure also makes it a trap for harmful substances :

• Environmental toxins such as dioxins, PCBs or pesticides are deposited in the lipid shell.
• Heavy metals (e.g. mercury, lead, cadmium) can bind to protein components.
• Endogenous toxins such as oxidized lipid residues or free radicals attach to LDL.

👉 LDL thus protects cells from direct damage caused by free, unbound toxins.

The perfect combination: LDL in the network

LDL does not act in isolation, but is part of a finely tuned system:

• HDL transports cholesterol and toxins back to the liver (“reverse cholesterol transport”).
• Antioxidants such as vitamins C and E stabilize LDL and prevent its oxidation.
• The liver and kidneys are the actual detoxification organs.
• The immune system uses LDL to neutralize bacterial endotoxins.

Thus, LDL becomes an important component of the body’s defense and detoxification .

LDL and heart disease: not a culprit, but an epiphenomenon

The classic hypothesis “high LDL = heart attack” is too simplistic – or simply wrong.

• Atherosclerosis is an inflammation : LDL only comes into play secondarily when the vessel wall is already inflamed.
• Oxidized LDL is problematic – not native LDL.
• Increased LDL can mean protection : The body produces more LDL to trap toxins and inflammatory products.
• Statins primarily act as anti-inflammatory agents , not primarily by lowering LDL.

👉 This makes it clear: LDL is not the cause of cardiovascular disease, but an epiphenomenon – a marker that points to the actual causes: chronic inflammation, oxidative stress and toxic exposure.

Solutions: Balance instead of combat

Instead of blindly lowering LDL, the goal is to support the body so that LDL remains stable, functional, and protective . Here are some natural approaches:

• Antioxidants
  Vitamin C, vitamin E, polyphenols from berries, green tea or dark chocolate protect LDL from oxidation.
• Curcuma longa (turmeric)
  The main active ingredient, curcumin, has strong anti-inflammatory and antioxidant properties. Studies show positive effects on vascular health and liver function.
• Olibanum (frankincense)
  Boswellic acids in frankincense have anti-inflammatory properties and support vascular and immune regulation.
• Colostrum (first milk)
  Rich in immunoglobulins, growth factors, and bioactive peptides, it strengthens the immune system and helps balance inflammatory processes.

👉 These measures neutralize free radicals, slow down chronic inflammation and relieve LDL so that it can continue to play its role as a transporter and toxin scavenger.

Conclusion: Time for a reassessment

LDL is not an enemy, but a versatile protection and transport vehicle.

• It absorbs toxins.
• It supplies the body with vital lipids and vitamins.
• It indicates when there are stresses in the body such as inflammation or oxidative damage.

Cardiovascular diseases are not caused by “too much LDL,” but by chronic inflammation, oxidative stress, and toxic influences .

In this context, LDL is not the cause but an epiphenomenon – a reflection of deeper processes that we need to understand and influence.